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Corrigendum to "Comprehensive assessment of estrogen receptor beta antibodies in cancer cell line models and tissue reveals critical limitations in reagent specificity" [Mol. Cell Endocrinol. 440 (2016) 138-150].

Molecular and cellular endocrinology

Nelson AW, Groen AJ, Miller JL, Warren AY, Holmes KA, Tarulli GA, Tilley WD, Katzenellenbogen BS, Hawse JR, Gnanapragasam VJ, Carroll JS.
PMID: 28183459
Mol Cell Endocrinol. 2017 Mar 05;443:175. doi: 10.1016/j.mce.2017.01.048.

No abstract available.

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Nelson AW, Groen AJ, Miller JL, et al. Corrigendum to "Comprehensive assessment of estrogen receptor beta antibodies in cancer cell line models and tissue reveals critical limitations in reagent specificity" [Mol. Cell Endocrinol. 440 (2016) 138-150]. Mol Cell Endocrinol. 2017;443:175doi: 10.1016/j.mce.2017.01.048.
Nelson, A. W., Groen, A. J., Miller, J. L., Warren, A. Y., Holmes, K. A., Tarulli, G. A., Tilley, W. D., Katzenellenbogen, B. S., Hawse, J. R., Gnanapragasam, V. J., & Carroll, J. S. (2017). Corrigendum to "Comprehensive assessment of estrogen receptor beta antibodies in cancer cell line models and tissue reveals critical limitations in reagent specificity" [Mol. Cell Endocrinol. 440 (2016) 138-150]. Molecular and cellular endocrinology, 443175. https://doi.org/10.1016/j.mce.2017.01.048
Nelson, A W, et al. "Corrigendum to "Comprehensive assessment of estrogen receptor beta antibodies in cancer cell line models and tissue reveals critical limitations in reagent specificity" [Mol. Cell Endocrinol. 440 (2016) 138-150]." Molecular and cellular endocrinology vol. 443 (2017): 175. doi: https://doi.org/10.1016/j.mce.2017.01.048
Nelson AW, Groen AJ, Miller JL, Warren AY, Holmes KA, Tarulli GA, Tilley WD, Katzenellenbogen BS, Hawse JR, Gnanapragasam VJ, Carroll JS. Corrigendum to "Comprehensive assessment of estrogen receptor beta antibodies in cancer cell line models and tissue reveals critical limitations in reagent specificity" [Mol. Cell Endocrinol. 440 (2016) 138-150]. Mol Cell Endocrinol. 2017 Mar 05;443:175. doi: 10.1016/j.mce.2017.01.048. PMID: 28183459; PMCID: PMC6854450.
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Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68].

Molecular and cellular endocrinology

Chen SC, Brooks R, Houskeeper J, Bremner SK, Dunlop J, Viollet B, Logan PJ, Salt IP, Ahmed SF, Yarwood SJ.
PMID: 28183460
Mol Cell Endocrinol. 2017 Mar 05;443:176. doi: 10.1016/j.mce.2017.01.049.

No abstract available.

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Chen SC, Brooks R, Houskeeper J, et al. Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68]. Mol Cell Endocrinol. 2017;443:176doi: 10.1016/j.mce.2017.01.049.
Chen, S. C., Brooks, R., Houskeeper, J., Bremner, S. K., Dunlop, J., Viollet, B., Logan, P. J., Salt, I. P., Ahmed, S. F., & Yarwood, S. J. (2017). Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68]. Molecular and cellular endocrinology, 443176. https://doi.org/10.1016/j.mce.2017.01.049
Chen, S C, et al. "Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68]." Molecular and cellular endocrinology vol. 443 (2017): 176. doi: https://doi.org/10.1016/j.mce.2017.01.049
Chen SC, Brooks R, Houskeeper J, Bremner SK, Dunlop J, Viollet B, Logan PJ, Salt IP, Ahmed SF, Yarwood SJ. Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68]. Mol Cell Endocrinol. 2017 Mar 05;443:176. doi: 10.1016/j.mce.2017.01.049. PMID: 28183460; PMCID: PMC6854454.
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Retraction notice to "TGF-β1 regulation of human AT1 receptor mRNA splice variants harboring exon 2".

Molecular and cellular endocrinology

Martin MM, Buckenberger JA, Knoell DL, Strauch AR, Elton TS.
PMID: 27519873
Mol Cell Endocrinol. 2016 Oct 15;434:288. doi: 10.1016/j.mce.2016.08.003.

No abstract available.

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Martin MM, Buckenberger JA, Knoell DL, et al. Retraction notice to "TGF-β1 regulation of human AT1 receptor mRNA splice variants harboring exon 2". Mol Cell Endocrinol. 2016;434:288doi: 10.1016/j.mce.2016.08.003.
Martin, M. M., Buckenberger, J. A., Knoell, D. L., Strauch, A. R., & Elton, T. S. (2016). Retraction notice to "TGF-β1 regulation of human AT1 receptor mRNA splice variants harboring exon 2". Molecular and cellular endocrinology, 434288. https://doi.org/10.1016/j.mce.2016.08.003
Martin, Mickey M, et al. "Retraction notice to "TGF-β1 regulation of human AT1 receptor mRNA splice variants harboring exon 2"." Molecular and cellular endocrinology vol. 434 (2016): 288. doi: https://doi.org/10.1016/j.mce.2016.08.003
Martin MM, Buckenberger JA, Knoell DL, Strauch AR, Elton TS. Retraction notice to "TGF-β1 regulation of human AT1 receptor mRNA splice variants harboring exon 2". Mol Cell Endocrinol. 2016 Oct 15;434:288. doi: 10.1016/j.mce.2016.08.003. PMID: 27519873.
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Corrigendum to "Differential gene regulation of GHSR signaling pathway in the arcuate nucleus and NPY neurons by fasting, diet-induced obesity, and 17β-estradiol" [Mol. Cell. Endocrinol. 157 (2016) 679-691].

Molecular and cellular endocrinology

Yasrebi A, Hsieh A, Mamounis KJ, Krumm EA, Yang JA, Magby J, Hu P, Roepke TA.
PMID: 27147476
Mol Cell Endocrinol. 2016 Jun 15;428:171-173. doi: 10.1016/j.mce.2016.04.017.

No abstract available.

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Yasrebi A, Hsieh A, Mamounis KJ, et al. Corrigendum to "Differential gene regulation of GHSR signaling pathway in the arcuate nucleus and NPY neurons by fasting, diet-induced obesity, and 17β-estradiol" [Mol. Cell. Endocrinol. 157 (2016) 679-691]. Mol Cell Endocrinol. 2016;428:171-173doi: 10.1016/j.mce.2016.04.017.
Yasrebi, A., Hsieh, A., Mamounis, K. J., Krumm, E. A., Yang, J. A., Magby, J., Hu, P., & Roepke, T. A. (2016). Corrigendum to "Differential gene regulation of GHSR signaling pathway in the arcuate nucleus and NPY neurons by fasting, diet-induced obesity, and 17β-estradiol" [Mol. Cell. Endocrinol. 157 (2016) 679-691]. Molecular and cellular endocrinology, 428171-173. https://doi.org/10.1016/j.mce.2016.04.017
Yasrebi, Ali, et al. "Corrigendum to "Differential gene regulation of GHSR signaling pathway in the arcuate nucleus and NPY neurons by fasting, diet-induced obesity, and 17β-estradiol" [Mol. Cell. Endocrinol. 157 (2016) 679-691]." Molecular and cellular endocrinology vol. 428 (2016): 171-173. doi: https://doi.org/10.1016/j.mce.2016.04.017
Yasrebi A, Hsieh A, Mamounis KJ, Krumm EA, Yang JA, Magby J, Hu P, Roepke TA. Corrigendum to "Differential gene regulation of GHSR signaling pathway in the arcuate nucleus and NPY neurons by fasting, diet-induced obesity, and 17β-estradiol" [Mol. Cell. Endocrinol. 157 (2016) 679-691]. Mol Cell Endocrinol. 2016 Jun 15;428:171-173. doi: 10.1016/j.mce.2016.04.017. PMID: 27147476.
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WITHDRAWN: Potential link between alpha 1 anti-trypsin and PAR-2 in the prevention of beta cell dysfunction(☆).

Molecular and cellular endocrinology

Kumar R, Balhuizen A, Soni A, Amisten S, Salehi A.
PMID: 21924322
Mol Cell Endocrinol. 2011 Sep 08; doi: 10.1016/j.mce.2011.08.040. Epub 2011 Sep 08.

This article has been withdrawn at the request of the authors. The Publisher apologizes for any inconvenience this may cause. The full Elsevier Policy on Article Withdrawal can be found at http://www.elsevier.com/locate/withdrawalpolicy.

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Kumar R, Balhuizen A, Soni A, et al. WITHDRAWN: Potential link between alpha 1 anti-trypsin and PAR-2 in the prevention of beta cell dysfunction(☆). Mol Cell Endocrinol. 2011;doi: 10.1016/j.mce.2011.08.040.
Kumar, R., Balhuizen, A., Soni, A., Amisten, S., & Salehi, A. (2011). WITHDRAWN: Potential link between alpha 1 anti-trypsin and PAR-2 in the prevention of beta cell dysfunction(☆). Molecular and cellular endocrinology, . https://doi.org/10.1016/j.mce.2011.08.040
Kumar, Rajesh, et al. "WITHDRAWN: Potential link between alpha 1 anti-trypsin and PAR-2 in the prevention of beta cell dysfunction(☆)." Molecular and cellular endocrinology vol. (2011). doi: https://doi.org/10.1016/j.mce.2011.08.040
Kumar R, Balhuizen A, Soni A, Amisten S, Salehi A. WITHDRAWN: Potential link between alpha 1 anti-trypsin and PAR-2 in the prevention of beta cell dysfunction(☆). Mol Cell Endocrinol. 2011 Sep 08; doi: 10.1016/j.mce.2011.08.040. Epub 2011 Sep 08. PMID: 21924322.
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Endocrine regulation of cancer stem cell compartments in breast tumors.

Molecular and cellular endocrinology

Zheng Y, Karnoub AE.
PMID: 34242715
Mol Cell Endocrinol. 2021 Sep 15;535:111374. doi: 10.1016/j.mce.2021.111374. Epub 2021 Jul 06.

Cancer cells within breast tumors exist within a hierarchy in which only a small and rare subset of cells is able to regenerate growths with the heterogeneity of the original tumor. These highly malignant cancer cells, which behave like...

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Zheng Y, Karnoub AE. Endocrine regulation of cancer stem cell compartments in breast tumors. Mol Cell Endocrinol. 2021;535:111374doi: 10.1016/j.mce.2021.111374.
Zheng, Y., & Karnoub, A. E. (2021). Endocrine regulation of cancer stem cell compartments in breast tumors. Molecular and cellular endocrinology, 535111374. https://doi.org/10.1016/j.mce.2021.111374
Zheng, Yurong, and Karnoub, Antoine E. "Endocrine regulation of cancer stem cell compartments in breast tumors." Molecular and cellular endocrinology vol. 535 (2021): 111374. doi: https://doi.org/10.1016/j.mce.2021.111374
Zheng Y, Karnoub AE. Endocrine regulation of cancer stem cell compartments in breast tumors. Mol Cell Endocrinol. 2021 Sep 15;535:111374. doi: 10.1016/j.mce.2021.111374. Epub 2021 Jul 06. PMID: 34242715.
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The roles of RUNX2 and osteoclasts in regulating expression of steroidogenic enzymes in castration-resistant prostate cancer cells.

Molecular and cellular endocrinology

Huang J, Hagberg Thulin M, Damber JE, Welén K.
PMID: 34216642
Mol Cell Endocrinol. 2021 Sep 15;535:111380. doi: 10.1016/j.mce.2021.111380. Epub 2021 Jul 01.

Intratumoral steroidogenesis is involved in development of castration-resistant prostate cancer (CRPC) as bone metastases. The osteoblast transcription factor RUNX2 influences steroidogenesis and is induced in CRPC cells by osteoblasts. This study investigates osteoclastic influence on RUNX2 in intratumoral steroidogenesis....

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Huang J, Hagberg Thulin M, Damber JE, et al. The roles of RUNX2 and osteoclasts in regulating expression of steroidogenic enzymes in castration-resistant prostate cancer cells. Mol Cell Endocrinol. 2021;535:111380doi: 10.1016/j.mce.2021.111380.
Huang, J., Hagberg Thulin, M., Damber, J. E., & Welén, K. (2021). The roles of RUNX2 and osteoclasts in regulating expression of steroidogenic enzymes in castration-resistant prostate cancer cells. Molecular and cellular endocrinology, 535111380. https://doi.org/10.1016/j.mce.2021.111380
Huang, Junchi, et al. "The roles of RUNX2 and osteoclasts in regulating expression of steroidogenic enzymes in castration-resistant prostate cancer cells." Molecular and cellular endocrinology vol. 535 (2021): 111380. doi: https://doi.org/10.1016/j.mce.2021.111380
Huang J, Hagberg Thulin M, Damber JE, Welén K. The roles of RUNX2 and osteoclasts in regulating expression of steroidogenic enzymes in castration-resistant prostate cancer cells. Mol Cell Endocrinol. 2021 Sep 15;535:111380. doi: 10.1016/j.mce.2021.111380. Epub 2021 Jul 01. PMID: 34216642.
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Carbonic-anhydrase IX expression is increased in thyroid cancer tissue and represents a potential therapeutic target to eradicate thyroid tumor-initiating cells.

Molecular and cellular endocrinology

Schmidt J, Oppermann E, Blaheta RA, Schreckenbach T, Lunger I, Rieger MA, Bechstein WO, Holzer K, Malkomes P.
PMID: 34216643
Mol Cell Endocrinol. 2021 Sep 15;535:111382. doi: 10.1016/j.mce.2021.111382. Epub 2021 Jun 30.

The expression of Carbonic-anhydrase IX (CAIX) in thyroid cancer (TC) subtypes was determined and its role in cancer cell growth and tumor-initiating cells (TICs) investigated. Immunohistochemistry in 114 TC patients revealed that CAIX expression was increased in tumor specimens...

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Schmidt J, Oppermann E, Blaheta RA, et al. Carbonic-anhydrase IX expression is increased in thyroid cancer tissue and represents a potential therapeutic target to eradicate thyroid tumor-initiating cells. Mol Cell Endocrinol. 2021;535:111382doi: 10.1016/j.mce.2021.111382.
Schmidt, J., Oppermann, E., Blaheta, R. A., Schreckenbach, T., Lunger, I., Rieger, M. A., Bechstein, W. O., Holzer, K., & Malkomes, P. (2021). Carbonic-anhydrase IX expression is increased in thyroid cancer tissue and represents a potential therapeutic target to eradicate thyroid tumor-initiating cells. Molecular and cellular endocrinology, 535111382. https://doi.org/10.1016/j.mce.2021.111382
Schmidt, Jennifer, et al. "Carbonic-anhydrase IX expression is increased in thyroid cancer tissue and represents a potential therapeutic target to eradicate thyroid tumor-initiating cells." Molecular and cellular endocrinology vol. 535 (2021): 111382. doi: https://doi.org/10.1016/j.mce.2021.111382
Schmidt J, Oppermann E, Blaheta RA, Schreckenbach T, Lunger I, Rieger MA, Bechstein WO, Holzer K, Malkomes P. Carbonic-anhydrase IX expression is increased in thyroid cancer tissue and represents a potential therapeutic target to eradicate thyroid tumor-initiating cells. Mol Cell Endocrinol. 2021 Sep 15;535:111382. doi: 10.1016/j.mce.2021.111382. Epub 2021 Jun 30. PMID: 34216643.
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Leukemia inhibitory factor prevents chicken follicular atresia through PI3K/AKT and Stat3 signaling pathways.

Molecular and cellular endocrinology

Dong J, Guo C, Zhou S, Zhao A, Li J, Mi Y, Zhang C.
PMID: 34990741
Mol Cell Endocrinol. 2022 Jan 04;543:111550. doi: 10.1016/j.mce.2021.111550. Epub 2022 Jan 04.

Autophagy of granulosa cell (GC) may be a supplementary mechanism involved in follicular atresia through cooperating with apoptosis. Leukemia inhibitory factor (LIF) has been shown to promote follicular growth, through the underlying molecular mechanisms remain unclear. Rapamycin, an autophagy...

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Dong J, Guo C, Zhou S, et al. Leukemia inhibitory factor prevents chicken follicular atresia through PI3K/AKT and Stat3 signaling pathways. Mol Cell Endocrinol. 2022;543:111550doi: 10.1016/j.mce.2021.111550.
Dong, J., Guo, C., Zhou, S., Zhao, A., Li, J., Mi, Y., & Zhang, C. (2022). Leukemia inhibitory factor prevents chicken follicular atresia through PI3K/AKT and Stat3 signaling pathways. Molecular and cellular endocrinology, 543111550. https://doi.org/10.1016/j.mce.2021.111550
Dong, Juan, et al. "Leukemia inhibitory factor prevents chicken follicular atresia through PI3K/AKT and Stat3 signaling pathways." Molecular and cellular endocrinology vol. 543 (2022): 111550. doi: https://doi.org/10.1016/j.mce.2021.111550
Dong J, Guo C, Zhou S, Zhao A, Li J, Mi Y, Zhang C. Leukemia inhibitory factor prevents chicken follicular atresia through PI3K/AKT and Stat3 signaling pathways. Mol Cell Endocrinol. 2022 Jan 04;543:111550. doi: 10.1016/j.mce.2021.111550. Epub 2022 Jan 04. PMID: 34990741.
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Non-genomic steroid signaling through the mineralocorticoid receptor: Involvement of a membrane-associated receptor?.

Molecular and cellular endocrinology

Karst H, den Boon FS, Vervoort N, Adrian M, Kapitein LC, Joëls M.
PMID: 34740745
Mol Cell Endocrinol. 2021 Nov 02;541:111501. doi: 10.1016/j.mce.2021.111501. Epub 2021 Nov 02.

Corticosteroid receptors in the mammalian brain mediate genomic as well as non-genomic actions. Although receptors mediating genomic actions were already cloned 35 years ago, it remains unclear whether the same molecules are responsible for the non-genomic actions or that...

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Karst H, den Boon FS, Vervoort N, et al. Non-genomic steroid signaling through the mineralocorticoid receptor: Involvement of a membrane-associated receptor?. Mol Cell Endocrinol. 2021;541:111501doi: 10.1016/j.mce.2021.111501.
Karst, H., den Boon, F. S., Vervoort, N., Adrian, M., Kapitein, L. C., & Joëls, M. (2021). Non-genomic steroid signaling through the mineralocorticoid receptor: Involvement of a membrane-associated receptor?. Molecular and cellular endocrinology, 541111501. https://doi.org/10.1016/j.mce.2021.111501
Karst, Henk, et al. "Non-genomic steroid signaling through the mineralocorticoid receptor: Involvement of a membrane-associated receptor?." Molecular and cellular endocrinology vol. 541 (2021): 111501. doi: https://doi.org/10.1016/j.mce.2021.111501
Karst H, den Boon FS, Vervoort N, Adrian M, Kapitein LC, Joëls M. Non-genomic steroid signaling through the mineralocorticoid receptor: Involvement of a membrane-associated receptor?. Mol Cell Endocrinol. 2021 Nov 02;541:111501. doi: 10.1016/j.mce.2021.111501. Epub 2021 Nov 02. PMID: 34740745.
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The role of fatty acids in Crohn's disease pathophysiology - An overview.

Molecular and cellular endocrinology

Piotrowska M, Binienda A, Fichna J.
PMID: 34480991
Mol Cell Endocrinol. 2021 Dec 01;538:111448. doi: 10.1016/j.mce.2021.111448. Epub 2021 Sep 01.

Crohn's disease (CD) is an inflammatory bowel disease (IBD) which is characterized by chronic and relapsing inflammation of the gastrointestinal (GI) tract. The etiology of CD is unknown, but factors such as epithelial barrier dysfunction, immune system imbalance, microbiota...

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Piotrowska M, Binienda A, Fichna J. The role of fatty acids in Crohn's disease pathophysiology - An overview. Mol Cell Endocrinol. 2021;538:111448doi: 10.1016/j.mce.2021.111448.
Piotrowska, M., Binienda, A., & Fichna, J. (2021). The role of fatty acids in Crohn's disease pathophysiology - An overview. Molecular and cellular endocrinology, 538111448. https://doi.org/10.1016/j.mce.2021.111448
Piotrowska, Marta, et al. "The role of fatty acids in Crohn's disease pathophysiology - An overview." Molecular and cellular endocrinology vol. 538 (2021): 111448. doi: https://doi.org/10.1016/j.mce.2021.111448
Piotrowska M, Binienda A, Fichna J. The role of fatty acids in Crohn's disease pathophysiology - An overview. Mol Cell Endocrinol. 2021 Dec 01;538:111448. doi: 10.1016/j.mce.2021.111448. Epub 2021 Sep 01. PMID: 34480991.
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Long noncoding RNA ZFAS1 suppresses osteogenic differentiation of bone marrow-derived mesenchymal stem cells by upregulating miR-499-EPHA5 axis.

Molecular and cellular endocrinology

Wu J, Lin T, Gao Y, Li X, Yang C, Zhang K, Wang C, Zhou X.
PMID: 34655661
Mol Cell Endocrinol. 2022 Jan 01;539:111490. doi: 10.1016/j.mce.2021.111490. Epub 2021 Oct 13.

Emerging evidence suggests that the shift between osteogenic and adipogenic differentiation of bone marrow-derived mesenchymal stem cells (BMSCs) determines bone mass. Our study was aimed at testing whether a long noncoding RNA called zinc finger antisense 1 (ZFAS1) participates...

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Wu J, Lin T, Gao Y, et al. Long noncoding RNA ZFAS1 suppresses osteogenic differentiation of bone marrow-derived mesenchymal stem cells by upregulating miR-499-EPHA5 axis. Mol Cell Endocrinol. 2021;539:111490doi: 10.1016/j.mce.2021.111490.
Wu, J., Lin, T., Gao, Y., Li, X., Yang, C., Zhang, K., Wang, C., & Zhou, X. (2022). Long noncoding RNA ZFAS1 suppresses osteogenic differentiation of bone marrow-derived mesenchymal stem cells by upregulating miR-499-EPHA5 axis. Molecular and cellular endocrinology, 539111490. https://doi.org/10.1016/j.mce.2021.111490
Wu, Jinhui, et al. "Long noncoding RNA ZFAS1 suppresses osteogenic differentiation of bone marrow-derived mesenchymal stem cells by upregulating miR-499-EPHA5 axis." Molecular and cellular endocrinology vol. 539 (2022): 111490. doi: https://doi.org/10.1016/j.mce.2021.111490
Wu J, Lin T, Gao Y, Li X, Yang C, Zhang K, Wang C, Zhou X. Long noncoding RNA ZFAS1 suppresses osteogenic differentiation of bone marrow-derived mesenchymal stem cells by upregulating miR-499-EPHA5 axis. Mol Cell Endocrinol. 2022 Jan 01;539:111490. doi: 10.1016/j.mce.2021.111490. Epub 2021 Oct 13. PMID: 34655661.
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